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Hepatic lipidosis
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Hepatic lipidosis is the end point of untreated gut stasis. Fat is broken down as an energy source and is broken down by beta-oxidation in the liver. A metabolic bottleneck occurs and ketoacidosis is the result. Affected rabbits die from liver and/or kidney failure. Disseminated intravascular coagulopathy may occur. Gastric ulceration is another feature of untreated gut stasis. This image shows the appearance of the liver and stomach of a rabbit that died from hepatic lipidosis: the liver is very pale and the dark areas on the stomach are ulcers. The primary problem was a dental spur.
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MACROSCOPIC CHANGES
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LIVER
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Liver lobe torsion
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This image shows the liver of a rabbit that had died from a liver lobe torsion. The caudal lobe has twisted around so the blood supply was cut off. The rest of the liver has fatty infiltration.
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MACROSCOPIC CHANGES
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LIVER
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Hepatic lipidosis
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This image shows the liver of a rabbit that died with hepatic lipidosis. She died a few hours after admission, despite intravenous fluids and other supportive treatment. She was ataxic and hypothermic with a low blood glucose (4.2 mmol/l) on admission. Her urine was acidic on a dipstick due to ketoacidosis. The rabbit had undergone radical dentistry at another practice 4 days earlier and had not eaten since she was discharged on the day of dentistry.
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MACROSCOPIC CHANGES
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LIVER
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Cystic liver
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Biliary cysts occasionally occur in rabbits. They are usually benign but may become calcified so they are visible on abdominal radiographs
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MACROSCOPIC CHANGES
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LIVER
