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Hepatic lipidosis
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This image shows the liver of a rabbit that died with hepatic lipidosis. She died a few hours after admission, despite intravenous fluids and other supportive treatment. She was ataxic and hypothermic with a low blood glucose (4.2 mmol/l) on admission. Her urine was acidic on a dipstick due to ketoacidosis. The rabbit had undergone radical dentistry at another practice 4 days earlier and had not eaten since she was discharged on the day of dentistry.
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MACROSCOPIC CHANGES
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LIVER
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Hepatic lipidosis
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Hepatic lipidosis is the end point of untreated gut stasis. Fat is broken down as an energy source and is broken down by beta-oxidation in the liver. A metabolic bottleneck occurs and ketoacidosis is the result. Affected rabbits die from liver and/or kidney failure. Disseminated intravascular coagulopathy may occur. Gastric ulceration is another feature of untreated gut stasis. This image shows the appearance of the liver and stomach of a rabbit that died from hepatic lipidosis: the liver is very pale and the dark areas on the stomach are ulcers. The primary problem was a dental spur.
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LIVER
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Cystic liver
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Biliary cysts occasionally occur in rabbits. They are usually benign but may become calcified so they are visible on abdominal radiographs
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Liver lobe torson
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This image shows the liver of a rabbit that died from a torsion of the caudate process of the caudal lobe of the liver (arrow). This is the most common part of the liver to be involved in liver lobe torsion in rabbits. The image shows its position behind the ribs on the right side of the abdomen
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MACROSCOPIC CHANGES
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LIVER
